P21

Cognitive Enhancement / Nootropic Peptideresearch

Also known as: P021, CNTF-derived peptide

A small synthetic peptide derived from ciliary neurotrophic factor (CNTF) that promotes neurogenesis and enhances cognitive function by inhibiting leukemia inhibitory factor (LIF) signaling.

Overview

P21 is a synthetic tetrapeptide developed by Dr. Khalid Iqbal at the New York State Institute for Basic Research in Developmental Disabilities. It is derived from the active region of ciliary neurotrophic factor (CNTF) but modified to be more stable and brain-penetrant. P21 promotes neurogenesis in the hippocampal dentate gyrus and reduces levels of hyperphosphorylated tau, making it a candidate for Alzheimer's disease and cognitive decline research. Unlike full-length CNTF, P21 lacks the LIF-activating domain, which means it promotes neurogenesis without the appetite-suppressing and weight-loss side effects of CNTF. In animal models, P21 has been shown to rescue cognitive deficits, increase dendritic spine density, and reduce neurodegeneration markers.

Mechanism of Action

P21 acts through several neuroplasticity-related pathways: (1) Mimics the neurotrophic activity of CNTF by activating similar downstream signaling; (2) Increases BDNF expression in the hippocampus, promoting neuronal survival and synaptic plasticity; (3) Promotes neurogenesis in the dentate gyrus of the hippocampus; (4) Reduces hyperphosphorylated tau protein levels, a hallmark of Alzheimer's pathology; (5) Inhibits LIF signaling selectively, avoiding the cachectic effects of full-length CNTF; (6) Enhances dendritic complexity and spine density in hippocampal neurons.

Molecular Formula

C25H40N6O6

Molecular Weight

520.62 g/mol

Sequence

Ac-DGGL-OH (Ac-Asp-Gly-Gly-Leu-OH)

Dosage Protocols

Dose Range

500mcg1000mcg

Frequency

Once daily

Route

intranasal

Cycle Length

4-12 weeks

Intranasal is the preferred route for direct CNS delivery. Reconstitute in bacteriostatic water. Experimental dosing based on animal study extrapolation.

Source: Community protocols derived from preclinical research

Side Effects

EffectSeverity
Nasal irritationmild
Headachemild
Mild fatiguemild
Vivid dreamsmild

Pros & Cons

Promotes actual neurogenesis (new neuron growth) in the hippocampus, not just neurotransmitter modulation

Reduces hyperphosphorylated tau — directly relevant to Alzheimer's disease pathology

Derived from CNTF but engineered to avoid appetite suppression and cachexia side effects

Small molecule size allows blood-brain barrier penetration and multiple administration routes

Very limited research — only a handful of animal studies from one research group

No human clinical trials or established safety data

Dosing protocols are entirely anecdotal and extrapolated from animal models

Limited availability and few reliable peptide suppliers carry it

Research Studies

Legal Status

Not FDA-approved. Available as a research chemical. No regulatory restrictions in most jurisdictions.

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